Sökresultat för Experimental hyperalgesia - Kliniska - ICH GCP
Stimulus-induced pain (hyperalgesia and allodynia) may result from sensitisation processes in the peripheral (primary hyperalgesia) or central (secondary hyperalgesia) nervous system. Se hela listan på academic.oup.com Increased pain sensitivity at a site of tissue damage is called primary hyperalgesia. Increased pain sensitivity in normal skin surrounding a site of tissue damage is called secondary hyperalgesia. Hyperalgesia was traditionally defined as the psychophysical correlate of sensitization (either peripheral or central) of the nociceptive system.
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Mech can be relased to NMDA receptors and endogenous pain modulating system eg depressed 25 Apr 2017 Along with the primary hyperalgesia that results from activation of peripheral nociceptors in response to injury, secondary hyperalgesia occurs 28 Oct 2019 whether sensations such as allodynia, hyperalgesia or dysaesthesia can be evoked. Corticosteroids (for example, when pain is secondary to nerve root of patients in palliative care wards versus those in general Allodynia is central sensitization pain response to stimuli that normally do not provoke pain. For example, for most people, stroking a cat is a pleasurable Hyperesthesia is a term that encompasses both allodynia and hyperalgesia. terminals from Aβ fibers and dendrites from lamina V projecting neurons. When a secondary neuron is depolarized it generates an action potential that is rel While this often means that allodynia and hyperalgesia seem to exist along the same continuum of stimuli on physical exam, there is still a clear difference in modalities.
Hyperalgesia and allodynia are frequent symptoms of disease and may be useful adaptations to protect vulnerable tissues. Enhanced sensitivity for pain may, however, persist long after the initial cause for pain has disappeared, then pain is no longer a symptom but rather a disease in its own right.
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Enhanced sensitivity for pain may, however, persist long after the initial cause for pain has disappeared, then pain is no longer a symptom but rather a disease in its own right. Whereas hyperalgesia to light touch (allodynia) is mediated by A-fibre low-threshold mechanoreceptors, hyperalgesia to punctate stimuli may be mediated by A- or C-fibre nociceptors.
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Anesthesia dolorosa. Causalgia. Central pain. Dysesthesia. Hyperalgesia*. Hyperesthesia.
2015-01-23 · Allodynia is different from hyperalgesia, which is an exaggerated response from a normally painful stimulus, although both can and often do co-exist.
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Secondary mechanical hyperalgesia to punctate mechanical stimuli and light touch 21 mA), punctate hyperalgesia (9 vs. 15 mA) and allodynia (15 vs. 18 mA). Nocicpetion vs pain?
2014-06-09 · For both A and B data from the central heat-conditioned and the adjacent skin area (P1) were pooled, since both areas displayed the same magnitude of change for heat-induced hyperalgesia and allodynia as well as in acetaminophen-related reduction of hyperalgesia or allodynia. * p<0.05, ** p<0.01, *** p<0.001 (t-test for repetitive heat-conditioning vs. unconditioned control).
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Fibromyalgia and chronic widespread pain - GUPEA
“allodynia“ and “pin prick“. Thermal hyperalgesia does not occur in the secondary zone.